Case report | DOI: https://doi.org/10.31579/2690-8794/317
*Corresponding Author: Suresh Kishanrao, Family Physician & Public Health Consultant Bengaluru, India.
Citation: Suresh Kishanrao, (2026), Engima of Pothole Jerk Reviving Brain-Dead Persons: Why do Routine Surgeries Go Wrong in India? Clinical Medical Reviews and Reports, 8(5); DOI:10.31579/2690-8794/317
Copyright: © 2026, Suresh Kishanrao. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Received: 03 April 2026 | Accepted: 14 April 2026 | Published: 01 May 2026
Keywords: Brain dead; Coma; revival; Vehicular jerk; brain death confirmation; Mortuary van; Ambulance
Indian media, Print, TV and social media take pleasure in airing sensational medical errors news which spread like wildfire every year. For example, on 11 March 2026 news title of “Bad Roads or Miracle? Brain-Dead Woman Revives after ambulance’s jerk on pothole. " Another news paper titled it as “A Pothole Saved 50-Year-Old Woman's Life After She Was declared Brain dead” Pothole Jolts 'Brain-Dead' Woman Back to Life In UP, a TV channel aired the even as” Pothole Jolts 'Brain-Dead' Woman Back to Life In UP”, yet another TV channel title it as “Pothole on NH4 did what Specialists had failed to do”, some others titled it as “a miracle”. And an Instagram news read “Pothole shock revives woman in Pilibhit, UP.
Medical science explains this phenomenon through a combination of potential misdiagnosis of confusing deep coma with true brain death in smaller settings where modern equipment’s and skill lock, the alleviation of physical pressure on the brain stem, & delayed response to targeted treatment for toxic poisoning. Often the family’s get the patient discharged against medical advice for financial reasons or ease of transporting live person than dead body.
Medically Brain death is defined as the irreversible cessation of all brain activity, including the brainstem. Based on medical science, any physical movement or a "vehicular jerk” cannot help recovering brain stem reflexes in a truly brain-dead person. Once brain death is confirmed by standard clinical exams, recovery is impossible, and if it happens, it clearly indicates an error on the part of the doctor or misunderstanding from the family. Cases where someone "recovers" after being declared brain-dead often involve, inaccurate or rushed diagnosis, where the patient was in a coma, a vegetative state, or suffering from temporary severe neurological impairment rather than true, total brain death.
Materials & Methods: This article is a review of media made sensational news in India of a woman getting revived by an ambulance jolt after hitting a pothole on the national highway and similar instances in the last 2 years.
Outcomes: All 3 cases reported between January 2024 & March 2026 were reported revived after a jolt of ambulances carrying them. They were not Brain death cases but cases of deep coma, misdiagnosed or were discharged with no hope of revival. Families found it easy to carry comatose patients than shifting dead bodies, as mortuary vans are not available.
The "enigma" of potholes in India seemingly reviving "brain-dead" individuals refers to a viral, heavily discussed incident on 11 March 2026 where a 50-year-old woman in Pilibhit, Uttar Pradesh declared clinically brain-dead, began breathing again after an ambulance hit a large pothole. Indian media, Print, TV and social media take pleasure in airing sensational medical errors news which spread like wildfire every year. For example, on 11 March 2026 episode was titled differently by different media channels as of “Bad Roads or Miracle? Brain-Dead Woman Revives after ambulance’s jerk on pothole. " Another news paper titled it as “A Pothole Saved 50-Year-Old Woman's Life After She Was declared Brain dead” Pothole Jolts 'Brain-Dead' Woman Back to Life In UP, a TV channel aired the even as” Pothole Jolts 'Brain-Dead' Woman Back to Life In UP”, yet another TV channel title it as “Pothole on NH4 did what Specialists had failed to do”, some others titled it as “a miracle” and an Instagram news read “Pothole shock revives woman in Pilibhit, UP. Medical professionals explained this phenomenon as a combination of potential misdiagnosis, the alleviation of physical pressure on the brain stem, and delayed response to targeted treatment for toxic poisoning [1].
The Comon question is why some routine assessments or interventions go wrong more often in India? There are no national statistics on post operative statistics, or medical errors. Indian Journal of Critical Care Medicine reported surgical complication to the tune of 27.5% compared to 19.8% globally [2]. Brain death is the permanent end of life, whereas a deep coma is a severe brain insult, where the patient is still alive and may, recover late fully or move into a vegetative state after recovery.
Medically Brain death is defined as the irreversible cessation of all brain activity, including the brainstem. Based on medical science, any physical movement or a "vehicular jerk” cannot help recovering brain stem reflexes in a truly brain-dead person. Once brain death is confirmed by standard clinical exams, recovery is impossible. Brain-dead patients can exhibit spontaneous movements or spinal reflexes (e.g., toe curling, limb movement) because the spinal cord can still react to stimulation, even though the brain is inactive. Spinal reflexes primarily facilitate and modulate breathing rather than controlling the fundamental respiratory rhythm, which is generated by the brainstem’s medulla oblongata and pons. While the brainstem creates the basic "drive to breathe," spinal circuits and reflexes refine this signal to ensure efficient muscle contraction and adjust to changing metabolic needs.
Where someone "recovers" after being declared brain-dead often involve inaccurate or rushed diagnosis, where the patient was in a coma, a vegetative state, or suffering from temporary severe neurological impairment rather than true, total brain death. 11 March 2026 incident was an error on the part of first hospital which rushed the diagnosis without conclusive proof, or the brain reflexes misinterpreted. It is also possible that the family misunderstood the explanation of the doctors for sheer convenience of shifting comatose individual than dead body as the revival was not assured.
This article is a review such media made sensational news in India in the last 2 years.
Case 1. Pothole on NH4 did what Specialists had failed to do: Vineeta Shukla a 50 years-old lady was discharged with discharge note saying “No signs of life” from a Bareli Hospital on 24th February 2026 as Brain stem reflexes had failed. Vineeta, a senior assistant in a government office in Pilibhit district, UP suddenly fainted on 22 February while doing some household work. She was taken to the State Govt. Medical College, from where she was referred to advance medical facility in Bareilly in a comatose status. On examination at Bareilly hospital i) her Glasgow Coma Scale was 3 points against a normal of 15 points ii) her eyes showed dilatation of pupils, an indication of brain death iii) no breathing iv) Observed constantly sinking signs of life in her. After a series of quick investigations, heavy neurotoxins were detected in her blood and lymphatic system. The consequent emergency treatment in ICU helped her make some recovery. But her condition reversed next day and on 24th family was told that there was “No signs of life “in her, and they can arrange to take her body back home if wanted! Disappointed and with grief, her husband Kuldeep organized an ambulance, called family to make arrangement for last rights and started journey towards Pilibhit. On their back home journey, her husband noticed that Vineeta started breathing after the ambulance struck a large pothole and the vehicle moved violently. Kuldeep then rushed her to a Private Neurocity Hospital, where the neurosurgeon went through a thorough inquiry about the patient’s condition admission with his counterparts in Barely hospital, and initiated treatment in ICU. Vineeta recovered her consciousness in about 4 days and started talking in a weeks’ time and then completely recovered to return home n Monday 9th March 2026.
Case 2. Darshan Singh Brar, Haryana: D S Brar an 80-year-old Indian man declared dead after being on a ventilator, in January 2024, came back to life when the ambulance transporting his body hit a deep pothole in Haryana. Darshan Singh Brar was being transported from Patiala to his home in Haryana, India. He had been on a ventilator for four days before doctors declared him dead While travelling, the ambulance experienced a severe jolt after hitting a pothole. The violent jolt caused him to move his hand, prompting his grandson to check for a pulse and find he was alive. The patient's grandson, who was in the ambulance, observed movement in the man's hand and felt a heartbeat. The family rushed him to a nearby hospital in Nising, where doctors confirmed he was alive, contrary to the previous death declaration, as reported by News18. Dr Netrapal from Rawal Hospital told NDTV, “We cannot say that the patient had died. When he was brought to us, he was breathing and had blood pressure as well as a pulse. He was still critical and in the ICU. The breathing is laborious, because he had an infection in his chest.”
Case 3. Declared dead man after heart attack moved fingers after the ambulance hit a pothole in Kolhapur, Maharashtra 03 January 2025: A 65-year-old man from Kolhapur, who was declared dead after a heart attack, came back to life when the ambulance carrying his body hit a pothole on the way to his funeral. The sudden jolt made his fingers move, leaving everyone shocked. His family rushed him back to the hospital, where doctors found a pulse and started treatment again. Turns out, the man had been alive the whole time - just deeply unconscious.
Brain death is the irreversible cessation of all brain functions, legally recognized as death, while a deep coma is a state of profound unconsciousness where some brain activity and reflexes may remain. Brain death involves a complete lack of brainstem reflexes, apnea (no breathing), and no response to pain, whereas coma patients may breathe spontaneously and have pupil reactions.
Key Differences Between Brain Death and Deep Coma
Brainstem Reflexes: In brain death, all brainstem reflexes are absent (fixed pupils, no gag/cough reflex, no response to oculovestibular testing). In deep coma, some reflexes may remain active.
| System’s Involved | Status in Brain Death | Status in Deep Coma |
| Respiratory Function | Brain-dead patients require a ventilator to breathe (apnea) | Comatose patients often can breathe on their own, even if unconscious. |
| Response to Pain | No response to intense stimulation | May show minimal, automatic reflex responses, |
| Recovery Potential | Permanent and irreversible | State of severe injury but, in some cases, may not be permanent. |
| Electroencephalogram (EEG) activity | Complete absence of electrical activity | Usually shows some level of brain wave activity |
Whereas in Brain Death case all the following signs must be present i) Signs of Permanent Unconsciousness like No response to environmental stimuli ii) Absent Brainstem Reflexes like Fixed pupils, no eye movement, no facial movement, no gag or cough reflex iii) Apnea: Total inability to breathe without a ventilator, confirmed by an apnea test (no breathing when removed from the ventilator as carbon dioxide levels rise), In Unconsciousness patients i) Eyes remain closed, patient cannot be aroused, ii) Preserved Reflexes like show pupillary light reaction or other reflexive movements like withdrawal from pain and Spontaneous Breathing meaning they are usually able to maintain breathing independently.
The process of confirming brain death globally: Brain death is globally confirmed as the irreversible cessation of all brain activity, including the brainstem, through a rigorous clinical protocol. It requires a known, irreversible cause of coma, absence of brainstem reflexes, and a positive apnea test demonstrating no breathing drive. Specialized ancillary tests like ECG, EEG, CT scans for intra cranial hemorrhage are vascular clotting, or angiography may support the diagnosis, but these are not available in smaller settings. Clinically key components of Brain Death Determination include: i) A known cause of coma (e.g., trauma, hemorrhage) that is irreversible ii) The patient has normal core temperature and blood pressure, and be free of sedatives or paralytic drugs iii) No response to painful stimuli indicating deep coma iv) Absent Brainstem Reflexes like No gag, cough, pupillary light reflex, or eye movements, v)
Ventilation is briefly stopped to confirm that the brainstem does not produce a respiratory response to high carbon dioxide levels. Ancillary Tests like confirming the absence of blood flow to the brain (4-vessel angiography, CT angiography) or lack of electrical activity (EEG, ECG) are used if clinical testing is inconclusive. Certification is done & signed off by two qualified physicians in India. Once the doctors explain the status and poor chances of revival, most families prefer to take home the patient than dead body due to socio-cultural beliefs and challenges of transporting dead bodies as most hospital do not give ambulance and don’t have “A hearse” or Funeral vehicle.
Neurological Explanations for the "Miracle"
Indian expert neurologists have dismissed the idea that a pothole can revive a truly "brain-dead" person but offered several explanations for this case: 1. First and foremost is misdiagnosis of Brain Death, the patient was likely in a deep, severe coma (GCS score of 3) rather than having fulfilled the strict, formal criteria for "brain stem death," which requires two separate sets of tests by a panel of doctors. 2. Physical Relief of Brain Herniation: The brain might have been herniating, putting pressure on the brainstem & stopping breathing. The violent jerk from the pothole may shift the brain, relieving that pressure iii) Sometimes in Toxin-Induced Coma, high neurotoxins from a venomous snake or insect bite, and the suspected "death" is reversible coma caused by toxins, already starting to recover when the jolt happened.
Contextual Factors: Potholes in India are a major public health menace, causing 9,438 deaths between 2020 and 2024, with UP reporting the highest fatalities. The incident has brought ironic attention to the dangerous state of roads in the region. Een in major cities like Bengaluru, Mumbai, Delhi roads are in bad shape due to heavy vehicular traffic and poor maintenance of the roads by Municipal authorities. Stories such as a "pothole revival miracle," spread rapidly as representing a rare, positive, and bizarre twist in the narrative of Indian infrastructure neglect.
Medical Explanation: Sudden coma (sudden loss of consciousness & irresponsiveness) in a person aged 50 or older is a medical emergency often caused by acute brain injury or insult cardiovascular events, or severe metabolic imbalances. The risk increases with age due to higher prevalence of underlying conditions such as diabetes, high blood pressure, and vascular disease. The top causes listed in India include:
1. Stroke or Intracranial Hemorrhage: A sudden bursting of a blood vessel (hemorrhage) or a blocked artery (ischemic stroke) in the brain can cause an immediate coma. Hemorrhagic strokes are more likely to cause rapid deterioration.
2. Acute Cardiovascular Event (Cardiac Arrest): A heart attack or arrhythmia can cause a sudden halt in blood flow to the brain, leading to anoxic brain injury (lack of oxygen) and immediate unconsciousness.
3. Traumatic Brain Injury (TBI): Especially relevant to older adults, as even trivial fall resulting in a head injury can cause immediate coma or an acute or subacute subdural hemorrhage (SDH-bleeding between the brain and skull).
4. Severe Metabolic Disturbances: a) Hypoglycemia/Hyperglycemia-which mean very low or very high blood sugar, in patients with diabetes b) Extreme sodium imbalance (hyponatremia/hypernatremia) or high calcium (hypercalcemia) c) Infections: Acute central nervous system infections like meningitis or encephalitis can lead to rapid swelling and coma d) Drug or Alcohol Overdose: Excessive doses of medications such as sedatives, opioids, or benzodiazepines can induce a rapid coma.
Risk Factors Specific to 50+ Age Group: Pre-existing Chronic Diseases like history of high blood pressure, diabetes, or previous heart disease, polypharmacy meaning taking multiple medications can increase the risk of accidental overdose or adverse interactions. A minor head banging or injury that might not affect a younger person can lead to bleeding inside the skull in older individuals.
Immediate Action/treatment: Treatment focuses on identifying the cause via brain imaging (CT/MRI) & blood tests, and reversing if possible (controlling blood sugar, stopping bleeding).
Revival of Respiration: Restoration of respiration involves techniques to restore breathing after failure, particularly from spinal cord injuries (SCI) or emergencies, using mechanical ventilation, diaphragm pacers, or nerve transfers. For acute situations, it involves opening the airway and artificial ventilation. Chronic cases, like high cervical SCI, use diaphragmatic pacing (phrenic nerve stimulation) to simulate normal breathing.
Spinal circuits receive respiratory drive from the brainstem and act as a "gain control" mechanism, strengthening or patterning the motor output to respiratory muscles, particularly the diaphragm and intercostals. Spinal interneurons, specifically propriospinal neurons, coordinate different respiratory muscle groups to ensure breathing is efficient based on body biomechanics. Proprioceptive feedback from respiratory muscles (muscle spindles and Golgi tendon organs) travels to the spinal cord, helping to modulate motor output, especially during high-demand conditions like exercise or hypercapnia (high CO2). Specific spinal cholinergic interneurons (V0C neurons) are activated during hypercapnic stress, increasing the amplitude of phrenic motor neuron output and augmenting the tidal volume. The pre-Bötzinger complex in the medulla is the essential generator for the breathing rhythm. Spinal Cord role is Facilitation. Propriospinal neurons shape this rhythm. Without input from the brainstem, the spinal circuitry alone cannot maintain normal respiration, but it can contribute to recovery of function following spinal cord injury (SCI).
Phrenic Nucleus (C3–C5): The motor neuron pool for the diaphragm receives direct inputs from propriospinal neurons that help facilitate diaphragmatic contraction. Thoracic Cord innervates intercostal and abdominal muscles, with spinal circuits coordinating thoracic motor pools. In summary, spinal reflexes act as a dynamic, modulatory system that allows the body to fine-tune the basic breathing rhythm generated in the brainstem, ensuring robust ventilation under various physiological demands.
Traumatic high cervical spinal cord injury (SCI) and SDH can result in a devastating loss of functional respiration, leaving patients permanently dependent on mechanical ventilation. Nerve transfer is a promising reinnervation strategy that has the potential to restore connectivity in paralyzed distal muscles. The spinal accessory nerve (SAN) remains functional in most cases after high cervical SCI and can serve as a donor to reinnervate the phrenic nerve (PN), thereby improving diaphragmatic function [5].
Sub-dural Hematoma: Subdural hematoma (SDH) management in India involves urgent diagnosis via CT/MRI, followed by conservative care (medicines, monitoring) for small, asymptomatic cases or surgical evacuation for symptomatic/acute cases. Burr hole drainage is the preferred, safest surgical technique for chronic SDH, while craniotomy is used for more complex cases. Surgical intervention for a subdural hematoma (SDH) is a critical, often lifesaving, procedure to relieve intracranial pressure, Acute SDH: Generally required if the clot is thicker than 1 cm or causes a midline brain shift of 5 mm. Chronic SDH: Recommended when the patient is symptomatic (headache, confusion, weakness) or if the hematoma is large. Typically, craniotomy for acute, solid clots or burr hole drainage for chronic, liquid blood are done in India. Common techniques include burr hole craniotomy, craniotomy (removing a skull flap), or, increasingly, minimally invasive middle meningeal artery (MMA) embolization [6].
a) Burr Hole Craniotomy: This is the primary treatment for chronic subdural hematomas (cSDH). A surgeon drills one or more small holes into the skull, inserts a tube, and drains blood, often leaving a drain for several days b) Craniotomy: Typically used for acute subdural hematomas, this more invasive procedure involves temporarily removing a larger section of the skull to access, evacuate thick blood clots, and address underlying damage c) Decompressive Craniectomy: Sometimes used if the brain is severely swollen and cannot accommodate the skull being replaced immediately d) Middle Meningeal Artery (MMA) Embolization: A minimally invasive, non-surgical procedure performed by neuro-interventionalists, often used to prevent recurrence of chronic subdural hematomas by blocking the artery feeding the hematoma e) Twist Drill Trephination: A bedside procedure sometimes used for patients too unstable for the operating room.
Standard electronic monitoring in the Intensive Care Unit (ICU) after subdural hematoma (SDH) surgery: Electronic monitoring devices focus on preventing secondary brain injury by:
1. Intracranial Pressure Monitoring:
a. Intracranial Pressure (ICP) Monitoring: Intraparenchymal Monitor (Bolt): A small probe placed into the brain tissue, commonly used for monitoring ICP as it is relatively low risk and allows for good waveform resolution. This is recommended if the patient is comatose, has signs of brain swelling, or a high risk of worsening after craniotomy as was in our first case.
b. electronic monitoring (EVD): A fluid-filled catheter inserted into the lateral ventricle, which is the gold standard for monitoring ICP, allowing for both continuous measurement and therapeutic drainage of cerebrospinal fluid (CSF).
c. Subdural Monitoring Device: Transducer-tipped catheters can be placed in the subdural space to measure ICP.
2. Neuro-ICU Standard Monitoring: Continuous EEG (cEEG) is used to detect non-convulsive seizures (NCS) or non-convulsive status epilepticus (NCSE), which are common following brain injury or Brain Tissue Oxygenation monitors specific brain tissue oxygen tension.
3. Neurological Checks: Regular assessment using the Glasgow Coma Scale (GCS) and Pupil checks are considered essential.
4. Hemodynamic Monitoring: Continuous Arterial Blood Pressure (A-line): Essential for calculating Cerebral Perfusion Pressure (CPP), which is (Mean Arterial Pressure - ICP).
5. Pulse Oximetry: To ensure adequate systemic oxygen saturation.
6. Transcranial Sonography (TGS): A bedside ultrasound tool used to monitor for recurrence of hematoma in chronic subdural hematoma (cSDH).
7. Near-Infrared Spectroscopy (NIRS): Noninvasive assessment of cerebral oxygenation
Complications and Outcomes: Complications can include recurrence, seizures, infection, or further bleeding. Recovery can take weeks to over a year. Acute SDH carries a high mortality rate due to underlying brain injury [6].
Medical management & Observations: Not all subdural hematomas require surgical intervention. Treating subdural hematoma without surgery is a viable option in following conditions i) Small and Stable Hematomas and not exerting significant pressure on the brain, ii) If a patient shows mild or no neurological impairments, such as dizziness or headaches iii) Chronic subdural hematomas which develop over weeks or months, the body often reabsorbs the blood naturally, iv) If CT scans determine that the hematoma is not shifting brain structures or if there’s no severe displacement v) In elderly patients or those with multiple health risks, non-surgical treatments is preferable to avoid complications from invasive procedures [7].
Medically Brain death is defined as the irreversible cessation of all brain activity, including the brainstem. Any physical movement or a "vehicular jerk” cannot help recovering brain stem reflexes in a truly brain-dead person. Though Brain-dead patients can exhibit spontaneous movements or spinal reflexes (e.g., toe curling, limb movement) as the spinal cord can still react to stimulation, even though the brain is inactive, revival is not possible. Such media reports need to be taken with a pinch of salt and not any miracles but can be judgmental errors either from a single doctor or families mis-understanding & other reasons
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